F - actin was labled and examined using fluorescence microscope.
標記F-actin,熒光顯微鏡下觀察.
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Even , upregulation of MOMA - 2 and downregulation of SM - actin were also detected in latter ( P < 0.05 ).
MOMA -2 表達增加,SM-actin表達降低 ( P<0.05 ).
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Methods ? ? - actin pseudogenes were found out with bioinformatics databases such as EMBL, PSEUDOGENE.
方法利用EMBL 、 PSEUDOGENE等生物信息數(shù)據(jù)庫,尋找β-actin的假基因.
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The gene expression of a - actin mRNA were determined by using the quantitative reverse transcription PCR technology.
利用半定量反轉(zhuǎn)錄聚合酶鏈式反應(yīng)(QRT-PCR)法測定骨骼肌α-actin基因表達.
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RT - PCR was used to determine GK and PEPCKgene expression ( corrected by ? ? - actin ) .
用RT -PCR 檢測GK和 PEPCK mRNA表達水平的變化.
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The role of actin filaments was disused in this paper.
本文討論了肌動蛋白微絲骨架在細胞融合過程中的作用.
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Upon discontinuing dexamethasone, cross - linking of the actin networks was reversible.
當終止地塞米松的作用后,小梁細胞內(nèi)的肌動蛋白交聯(lián)是可逆的.
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And the chromosomes and organelles were successively connected with actin filaments during cytomixis.
在細胞融合過程中,有微絲骨架纖維與穿壁轉(zhuǎn)移的染色質(zhì)和細胞器相連.
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A large portion of the cytoskeleton consists of threadlike microfilaments composed mainly of the contractile actin.
細胞骨架大部分由微絲組成,微絲主要由可收縮的肌動蛋白組成.
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Cells are likely to be deteched when the quantity of actin filament decreases.
細胞中肌動蛋白纖維含量減少時,細胞易從基質(zhì)上脫落.
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The Molecular Study of Nuclear Actin Involving in the Regulation of Gene Transcription.
細胞核肌動蛋白與RNA聚合酶關(guān)系的研究.
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Conclusion the decreased actin activity is one of the reasons the decline of cardiac systolic potential.
結(jié)論:肌動蛋白活性降低是引起收心肌收縮力降低的原因之一.
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In plant cells, actin cytoskeleton plays important roles in response to biotic and abiotic stresses.
植物細胞對外界生物或非生物的脅迫做出反應(yīng)時, 微絲骨架起了重要的作用.
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Microtubules are shown in green, actin is in red and mitotic chromosomes are colored blue.
綠色為微絲, 紅色為肌動蛋白,藍色為染色質(zhì),中心點復(fù)制成兩個.
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Disrupted actin cytoskeleton could be recovered by removal of the VD - toxin before the nucleoli were dyed.
在核仁能夠看見以前,即核仁開始著色以前將毒素除去,可以使已經(jīng)破壞的周質(zhì)微絲骨架恢復(fù)過來.
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