Methods: 33 post - thoracotomy patients were divided into DCA, PCEA and PCIA groups.
方法: 將開胸術(shù)后病人33例隨機(jī)平均分入DCA組 、 PCEA組、PCIA組.
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DCA up - regulated the expression of AP - l ( c - Jun c - Fos ) and COX - 2 in a dose dependent manner.
DCA能上調(diào)AP -1 ( c -Jun 、c-Fos ) 蛋白及COX -2 蛋白表達(dá),呈濃度依賴性.
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In this case, LSS - DCA aligned the sequences with the same way as ClustalW.
在這種情況下, LSS-DCA的策略就不起作用,只采用ClustalW對(duì)齊序列.
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The kinetics and mechanism of exciplex formation of 9,10 - dicyanoanthracene ( DCA ) by acenaphthenone ( ANO ) have been studied.
研究了苊酮 ( ANO ) 對(duì)9,10-二氰蒽 ( DCA ) 的熒光猝滅與激基絡(luò)合物形成的動(dòng)力學(xué)與機(jī)理.
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Results: 200 μ M DCA induced cell proliferation and COX - 2 mRNA transcription.
結(jié)果: 200μMDCA可以 促進(jìn)HT-29 細(xì)胞的增殖,并誘導(dǎo)COX -2 mRNA的轉(zhuǎn)錄、COX-2蛋白的表達(dá)及PGE2的合成.
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