The levels of CD 3, CD 4 were markedly increased after treatment of CHM.
中藥治療后CD3 、 CD4明顯增高.
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Immunohistochemical analysis revealed a predominant CD 4 + T cell infiltration.
免疫組化示CD4+T細(xì)胞為主的淋巴細(xì)胞浸潤(rùn).
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Conclusion: CD 4 + T cell may the key role in the pathogenesis of ACD reaction.
結(jié)論: ACD反應(yīng)CD4+T細(xì)胞的主要參與.
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Conclusion: Aire gene does not affect the production of CD 4~ + CD 25~ + regulatory T cells.
結(jié)論: 結(jié)果表明Aire基因不影響CD4+CD25+調(diào)節(jié)性T細(xì)胞的產(chǎn)生.
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Conclusion BioA can inhibit the activity of HIV - 1 and the early activation of human CD 4 + lymphocytes.
L-1達(dá)到最大抑制率,能使活化率從(60.42±0.52)%降低到(19.70±0.38)%.結(jié)論BioA具有 抗HIV -1 介導(dǎo)的細(xì)胞融合和HIV-1復(fù)制的活性及抑制CD4+淋巴細(xì)胞早期活化作用.
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Active CD 4 + and CD 8 + T cells have antitumor immunity and therapy functions.
活化CD4+和CD8+T細(xì)胞,同時(shí)產(chǎn)生抗腫瘤免疫和治療作用.
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Objective : To investigate the immunoregulatory mechanism of CD 4 + CD 25 + regulatory T cells during the early phase of P . yoelii infection.
目的探討CD4+CD25+調(diào)節(jié)性T細(xì)胞在約氏瘧原蟲(chóng)感染早期的免疫調(diào)節(jié)機(jī)制.
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ObjectiveTo investigate the effects of Bccillus Calmette Guerin ( BCG ) vaccine on anti - tuberculosis and CD 4 + CD 8 + double positive ( DP ) T lymphocytes.
目的探討卡介苗 ( BCG ) 對(duì)大鼠結(jié)核感染及CD4+CD8+雙陽(yáng) ( DP ) T細(xì)胞的影響.
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After a rise in CD 4 - cells under ART , indications should be reconsidered, some vaccinatinations repeated.
抗逆轉(zhuǎn)錄病毒治療使CD4細(xì)胞數(shù)升高后, 應(yīng)該考慮接種指示, 并重復(fù)接種免疫.
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CD 3 +, CD 4 +, CD 8 + T lymphocytes in peripheral blood decreased apparently in patients with SARS.
外周血CD3~+ 、 CD4~+ 、 CD8~+明顯降低.
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Objective To observe autophagy in peripheral blood CD 4 + T lymphocyte of asthma patient.
目的觀察分離培養(yǎng)的哮喘病人外周血CD4+T淋巴細(xì)胞的自噬現(xiàn)象.
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The percentage of CD 4 + CD 25 + in asthma mouse treated with BCG ( 14.4±2.75 ) % was higher than asthma mouse ( P < 0.05 ).
而B(niǎo)CG干預(yù)組CD4+CD25+Treg百分比為(14.4±2.75 ) %,較哮喘組明顯上升 ( P<0.05 ).
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Objectives To explore the clinical features and treatment of idiopathic CD 4 + T - lymphocytopenia.
目的分析特發(fā)性CD4+T淋巴細(xì)胞減少癥的臨床特點(diǎn),并探討其發(fā)病機(jī)制和治療.
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Human Immunodeficiency Virus ( HIV ) produces a protein that prevents the activation of CD 4 + T cells.
人免疫缺陷病毒 ( HIV ) 可產(chǎn)生一種蛋白質(zhì),阻止CD4+T細(xì)胞的激活.
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