Induced by FasL + CD 34 ++ DNR ? FasL + CD 34 ++ Arac - that was 13.4 % ±1.0 % ( P < 0.05 ) ? 17.9 % ±1.3 % ( P < 0.01 ).
FasL +CD34++DNR 或FasL+CD34++Arac 作用后,細(xì)胞凋亡率分別為13.4%±1.0% ( P<0.05 ) 、17.95%±1.3% ( P<0.01 ).
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Aim : To explore the sensitivity of survivin - positive primary acute leukemia ( AL ) cells to aclacinomycin ( Acla ) and cytarabine ( AraC ).
目的探討survivin基因表達(dá)的原代急性白血病 ( AL ) 細(xì)胞對(duì)阿克拉霉素 ( Acla ) 聯(lián)合阿糖胞苷 ( AraC ) 的敏感性.
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Conclusion : It'suggests that the survivin - positive AL cells are probably sensitive to AcLa and AraC.
結(jié)論survivin基因陽(yáng)性表達(dá)的原代AL細(xì)胞對(duì)AA較敏感.
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Effects of antineoplasties drugs ( CY, 5 Fu, MTX, AraC and MMC ) on lipid peroxidation in mice were studided.
研究抗腫瘤藥甲氨蝶呤 、 阿糖胞苷 、 環(huán)磷酰胺 、 5—氟尿嘧 啶 和絲裂霉素C對(duì)小鼠脂質(zhì)過(guò)氧化作用的影響、結(jié)果顯示:5種抗腫瘤藥使小鼠血清和肝、腦中MDA含量 顯著升高.
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